Страница 182

22 июля 2026, 01:47

28 Moore, L. D., Le, T., Fan, G. 2013. “DNA Methylation and Its Basic Function”. Neuropsychopharmacology 38:23–38, https://doi.org/10.1038/npp.2012.112.

29 Herb, B. R., et al. 2012. “Reversible Switching between Epigenetic States in Honeybee Behavioral Subcastes”. Nature Neuroscience 15:1371–73, https://doi.org/10.1038/

30 Horvath, S. 2013. “DNA Methylation Age of Human Tissues and Cell Types”. Genome Biology 14: R115, https://doi.org/10.1186/gb-2013-14-10-r115.

31 Ha

32 Liu, Z., et al. 2020. “Under lying Features of Epigenetic Aging Clocks In Vivo and In Vitro”. Aging Cell 19: e13229, https://doi.org/10.1111/acel.13229; Levine, M. E. 2020. “Assessment of Epigenetic Clocks as Biomarkers of Aging in Basic and Population Research”. Journals of gerontology, Series A, Biological Sciences and Medical Sciences 75:463–65, https://doi.org/10.1093/gerona/glaa021; Schmitz, L. L., et al. 2021. “The Socioeconomic Gradient in Epigenetic Ageing Clocks: Evidence from the Multi-ethnic Study of Atherosclerosis and the Health and Retirement Study”. Epigenetics 17:589–611, опубликовaно в электронном виде до выходa в печaть 6 июля 2021 г., https://doi.org/10.1080/15592294.2021.1939479.

33 Levine, M. E., et al. 2016. “Menopause Accelerates Biological Aging”. Proceedings of the National Academy of Sciences of the USA 113:9327–32, https://doi.org/10.1073/pnas.1604558113.

34 Buckley, M. T., et al. 2022. “Cell Type-Specific Aging Clocks to Quantify Aging and Rejuvenation in Regenerative Regions of the Brain”. bioRxiv, 2022.01.10.475747, https://doi.org/10.1101/2022.01.10.475747.

35 Olins, D. E., Olins, A. L. 1978. “Nucleosomes: The Structural Quantum in Chromosomes”. American Scientist 66:704–11.

36 Farrelly, L. A., et al. 2019. “Histone Serotonylation Is a Permissive Modification that Enhances TFIID Binding to H3K4me3”. Nature 567:535–39, https://doi.org/10.1038/s41586-019-1024-7.

37 Ma, Z., et al. 2018. “Epigenetic Drift of H3K27me3 in Aging Links Glycolysis to Healthy Longevity in Drosophila”. eLife 7: e35368, https://doi.org/10.7554/eLife.35368.

38 Cheung, P., et al. 2018. “Single-Cell Chromatin Modification Profiling Reveals Increased Epigenetic Variations with Aging”. Cell 173:1385–97.e14, https://doi.org/10.1016/j.cell.2018.03.079.

39 Benayoun, B. A., et al. 2019. “Remodeling of Epigenome and Transcriptome Landscapes with Aging in Mice Reveals Widespread Induction of Inflammatory Responses”. Genome Research 29:697–709, https://doi.org/10.1101/gr.240093.118.

40 Greer, E. L., et al. 2010. “Members of the H3K4 Trimethylation Complex Regulate Lifespan in a Germline-Dependent Ma

41 Maures, T. J., Greer, E. L., Hauswirth, A. G., Brunet, A. 2011. “The H3K27 Demethylase UTX-1 Regulates C. elegans Lifespan in a Germline-Independent, Insulin-Dependent Ma

42 Guillermo, A. R. R., et al. 2021. “H3K27 Modifiers Regulate Lifespan in C. elegans in a Context-Dependent Ma

43 Greer, E. L., et al. 2011. “Transgenerational Epigenetic Inheritance of Longevity in Caenorhabditis elegans”. Nature 479:365–71, https://doi.org/10.1038/nature10572.

44 Han, S., et al. 2017. “Mono-unsaturated Fatty Acids Link H3K4me3 Modifiers to C. elegans Lifespan”. Nature 544:185–90, https://doi.org/10.1038/nature21686.

45 Lee, T. W., David, H. S., Engstrom, A. K., Carpenter, B. S., Katz, D. J. 2019. “Repressive H3K9me2 Protects Lifespan against the Transgenerational Burden of COMPASS Activity in C. elegans”. eLife 8: e48498, https://doi.org/10.7554/eLife.48498.

46 Liu, Z. C., Ambros, V. 1989. “Heterochronic Genes Control the Stage-Specific Initiation and Expression of the Dauer Larva Developmental Program in Caenorhabditis elegans”. Genes and Development 3:2039–49, https://doi.org/10.1101/gad.3.12b.2039; Lee, R. C., Ambros, V. 2001. “An Extensive Class of Small RNAs in Caenorhabditis elegans”. Science 294:862–64, https://doi.org/10.1126/science.1065329.

47 Zia, A., Farkhondeh, T., Sahebdel, F., Pourbagher-Shahri, A. M., Samarghandian, S. 2021. “Key miRNAs in Modulating Aging and Longevity: A Focus on Signaling Pathways and Cellular Targets”. Current Molecular Pharmacology 15:736–62, https://doi.org/10.2174/1874467214666210917141541; Kinser, H. E., Pincus, Z. 2020. “MicroRNAs as Modulators of Longevity and the Aging Process”. Human Genetics 139:291–308, https://doi.org/10.1007/s00439-019-02046-0; Boehm, M., Slack, F. 2005. “A Developmental Timing MicroRNA and Its Target Regulate Life Span in C. elegans”. Science 310:1954–57, https://doi.org/10.1126/science.1115596.

48 Zia et al. 2021; Kinser, Pincus 2020; Boehm, Slack 2005.

49 Baugh, L. R., Hu, P. J. 2020. “Starvation Responses throughout the Caenorhabditis elegans Life Cycle”. Genetics 216:837–78, https://doi.org/10.1534/genetics.120.303565.

50 Angelo, G., Van Gilst, M. R. 2009. “Starvation Protects Germline Stem Cells and Extends Reproductive Longevity in C. elegans”. Science 326:954–58, https://doi.org/10.1126/science.1178343.

51 Jobson, M. A., et al. 2011. “Transgenerational Effects of Early Life Starvation on Growth, Reproduction, and Stress Resistance in Caenorhabditis elegans”. Genetics 201:201–12, https://doi.org/10.1534/genetics.115.178699; Greer et al. 2011.

52 Rechavi, O., et al. 2014. “Starvation-Induced Transgenerational Inheritance of Small RNAs in C. elegans”. Cell 158:277–87, https://doi.org/10.1016/j.cell.2014.06.020.

Пока нет комментариев. Авторизуйтесь, чтобы оставить свой отзыв первым!